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Gugliucci Lectures Stats
Can you name the Gugliucci Lectures ?
By
touroyeah
15m
37 Questions
18 Plays
18 Plays
18 Plays
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Score
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x
% Correct
A or B: N-
acetyl glucosamine
A
100%
A fetus synthesizes heme in this organ
liver
94.4%
A clinical sign characterized by yellow skin, sclera, and mucosa. Bilirubin is 3X above normal.
Jaundice
94.4%
Bilirubin is conjugated with glucuronic acid in this organ
liver
88.9%
RBC synthesis takes how many days
7
88.9%
A or B: Galactose
B
88.9%
Endothelial injury, abnormal blood flow, and hypercoagulability make up_
_
_
_
_
_
_
_
_
_
_
_
_
_
_
_
triad
Virchow's
88.9%
Intrinsic or Extrinsic: Most physiologically relevant in driving coagulation after vascular damage.
extrinsic
88.9%
Where does heme synthesis occur?
Erythroblast in Bone Marrow
83.3%
Unconjugated bilirubin is transported through the blood with this protein
albumin
83.3%
A,B,O are alleles of the same gene on a single locus on this chromosome
9
83.3%
A presence of this protein means the person is Rh+
D protein
83.3%
Degranulates, changes shape, and puts out 3 receptors when activated.
platelets
83.3%
Exposed when the vessel has a lesion. GPIb binds to this.
vWF
83.3%
Treatment monitored with APTT
Heparin
83.3%
Process that breaks down the clot and has enzymatic activity of plasmin to break down fibrin and interfere with its polymerization
Fibrinolysis
83.3%
Fetal blood gets in touch with the mother's blood when this detaches.
placenta
77.8%
Takes up RBC after 120 days
Spleen macrophage
72.2%
Name a granule released by platelets
ADP or TXA2
72.2%
x
x
% Correct
A protease that cleaves fibrinogen into fibrin. Other functions include activation of endothelium to generate tPA, NO, PGI2, and PDGF mediators.
Thrombin
72.2%
Used to cover the baby's Rh+ RBC
RhoGAM
66.7%
Where coagulation factors are synthesized
liver
66.7%
Ratio of the patient's PT to the mean PT for a group of normal patients
INR
66.7%
A disease where patients are photosensitive and the most common type of error in heme synthesis.
Porphyria Cutanea Tarda
61.1%
Coagulation cascade includes 1) tissue factor 2) phospholipid complex expression 3) thrombin activation and 4)_
_
_
_
_
_
_
_
_
_
fibrin polymerization
55.6%
Most important of the plasminogen activators
tPA
55.6%
Released by endothelial cells as anti-
coagulant.
Prostacyclin
50%
Accumulation of ALA and porphobillinogen in the urine and no photosensitization characterizes this disease caused by errors in heme synthesis.
Acute intermittent porphyria
44.4%
Inhibits clotting by proteolytic cleavage of factor Va and VIIIa
Protein C
44.4%
Reduced heme in a linear chain and is green in color
bilverdin
38.9%
20X Bilirubin levels that goes to lipid membranes and accumulate in basal ganglia result into this neurological disorder in neonates.
Kernicterus
38.9%
Treatment monitored with PT
Coumadin
38.9%
Activated by heparin and inactivates thrombin, Xa, and IXa
AT III
33.3%
O allele codes for the inactive form of this enzyme
glycosyl transferase
22.2%
Neonatal jaundice has low activity of this hepatic enzyme, which reaches adult level in 4 weeks.
bilirubin gluconyltransferase
16.7%
Deficiency in this receptor results in Glanzmann thrombastenia
GpIIb-IIIa
16.7%
Deficiency in GPIb results into this syndrome
Bernard-Soulier syndrome
11.1%
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