Science Quiz / Gugliucci Lectures

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Can you name the Gugliucci Lectures ?

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Neonatal jaundice has low activity of this hepatic enzyme, which reaches adult level in 4 weeks.
Released by endothelial cells as anti-coagulant.
O allele codes for the inactive form of this enzyme
Accumulation of ALA and porphobillinogen in the urine and no photosensitization characterizes this disease caused by errors in heme synthesis.
Coagulation cascade includes 1) tissue factor 2) phospholipid complex expression 3) thrombin activation and 4)__________
A presence of this protein means the person is Rh+
Exposed when the vessel has a lesion. GPIb binds to this.
Deficiency in this receptor results in Glanzmann thrombastenia
Takes up RBC after 120 days
Unconjugated bilirubin is transported through the blood with this protein
A fetus synthesizes heme in this organ
A disease where patients are photosensitive and the most common type of error in heme synthesis.
Where does heme synthesis occur?
Deficiency in GPIb results into this syndrome
Process that breaks down the clot and has enzymatic activity of plasmin to break down fibrin and interfere with its polymerization
A protease that cleaves fibrinogen into fibrin. Other functions include activation of endothelium to generate tPA, NO, PGI2, and PDGF mediators.
Endothelial injury, abnormal blood flow, and hypercoagulability make up________________ triad
Degranulates, changes shape, and puts out 3 receptors when activated.
Activated by heparin and inactivates thrombin, Xa, and IXa
xxx
Name a granule released by platelets
Used to cover the baby's Rh+ RBC
A or B: Galactose
Reduced heme in a linear chain and is green in color
Ratio of the patient's PT to the mean PT for a group of normal patients
Most important of the plasminogen activators
Intrinsic or Extrinsic: Most physiologically relevant in driving coagulation after vascular damage.
RBC synthesis takes how many days
Where coagulation factors are synthesized
Fetal blood gets in touch with the mother's blood when this detaches.
Inhibits clotting by proteolytic cleavage of factor Va and VIIIa
20X Bilirubin levels that goes to lipid membranes and accumulate in basal ganglia result into this neurological disorder in neonates.
A,B,O are alleles of the same gene on a single locus on this chromosome
Treatment monitored with APTT
Bilirubin is conjugated with glucuronic acid in this organ
Treatment monitored with PT
A clinical sign characterized by yellow skin, sclera, and mucosa. Bilirubin is 3X above normal.
A or B: N-acetyl glucosamine

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