Science Quiz / USMLE Renal Pathologies

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Can you name the USMLE Renal Pathologies?

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Associated with gout, not seen on Xray
Painless hematuria, caused by cyclophosphamide, phenacetin, aniline dyes
Increased urine pH, acidosis, nonfunctional collecting tubule cells
Bilateral outflow tract obstruction, increased FENa
ureteric bifurcation or doubling, increased UTI risk
Painless hematuria, caused by chronic S. haematobium infection
Found in pelvis, trapped by IMA
Wire loop capillaries, caused by SLE
Flank pain, eosinophilic casts, thyroidization of tubules
Coca cola urine, subepithelial humps, hypercellular glomeruli
Hematuria, basket weave GBM, eye/ear pathologies
Toxic necrosis of tubular epithelial cells in PCT
Sporadic defect, nonfunctional kidney made of cysts, connective tissue
Mesangial proliferation, IgA deposits on IF
Crescents, MPO-ANCA, no IF deposits
Diffuse GBM thickening, spike and dome pattern on EM
eosinophilic nodular glomerulosclerosis (Kimmelstiel Wilson)
Flank pain, WBC casts, neutrophils in interstitium
Defective PCT bicarbonate reuptake, hypokalemia, acidosis
Envelope crystals, caused by increased urine pH
Upper/Lower respiratory symptoms, crescents, PR3-ANCA, no IF deposits
Common in kids, effaced podocytes, responds to steroids
Hematuria, arthralgias, rash, GI distress
Incomplete bladder emptying, leakage with overfilling
Linear IF deposits, crescents, anti-GBM antibodies
Tram track deposits, 2nd type due to C3 nephritic factor
HintAnswer
Sloughing of necrotic papilla, caused by diabetes, sickle cell, NSAIDs
Mass with central scar, eosinophilic cells, no perinuclear clearing
Suprapubic pain, dysuria, positive urine nitrites
Secondary hyperPTH due to decreased Vitamin D production
Cortical infarction caused by DIC from obstetric issues
Hyperkalemia, decreased urine pH, hypoaldosteronism
Global defect with PCT resorption
Forms staghorn calculi, caused by urease positive bacteria
Cysts in cortex and medulla, seen with aneurysms and mitral valve prolapse
Cystic dilation of collecting ducts, can cause oligohydramnios
Overactive bladder, leakage with urge to void immediately
Outlet incompetence, leakage with increased abdominal pressure
Wedge shaped crystals, caused by hypocitraturia
Malignancy of PCT cells, clear cytoplasm
Decreased renal blood flow, increased BUN/creatinine ratio
Inherited defect in Na/K/2Cl transporter
Inherited defect in NaCl transporter in DCT
Congo red stain, apple green birefringence
Eosinophils in urine, drug hypersensitivity reaction
Inherited defect, constituitively activates ENaC channel
Caused by PCT amino acid transporter defect, seen in children
Found in children, contains embryonic tissue, found in Beckwith-Wiedemmann
Oligohydramnios leading to lung hypoplasia
Tubulointerstitial fibrosis, unable to concentrate urine, poor prognosis
Decreased renal blood flow causes tubular ischemia
Segmental sclerosis of glomeruli, foot process effacement, common in AA/hispanics

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