Science Quiz / USMLE Renal Pathologies

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Can you name the USMLE Renal Pathologies?

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Mass with central scar, eosinophilic cells, no perinuclear clearing
Painless hematuria, caused by chronic S. haematobium infection
Suprapubic pain, dysuria, positive urine nitrites
Hematuria, basket weave GBM, eye/ear pathologies
Incomplete bladder emptying, leakage with overfilling
Hyperkalemia, decreased urine pH, hypoaldosteronism
Associated with gout, not seen on Xray
eosinophilic nodular glomerulosclerosis (Kimmelstiel Wilson)
Overactive bladder, leakage with urge to void immediately
Toxic necrosis of tubular epithelial cells in PCT
Bilateral outflow tract obstruction, increased FENa
Found in pelvis, trapped by IMA
ureteric bifurcation or doubling, increased UTI risk
Painless hematuria, caused by cyclophosphamide, phenacetin, aniline dyes
Congo red stain, apple green birefringence
Decreased renal blood flow causes tubular ischemia
Oligohydramnios leading to lung hypoplasia
Sloughing of necrotic papilla, caused by diabetes, sickle cell, NSAIDs
Flank pain, WBC casts, neutrophils in interstitium
Outlet incompetence, leakage with increased abdominal pressure
Tubulointerstitial fibrosis, unable to concentrate urine, poor prognosis
Decreased renal blood flow, increased BUN/creatinine ratio
Inherited defect, constituitively activates ENaC channel
Diffuse GBM thickening, spike and dome pattern on EM
Global defect with PCT resorption
Coca cola urine, subepithelial humps, hypercellular glomeruli
Flank pain, eosinophilic casts, thyroidization of tubules
Sporadic defect, nonfunctional kidney made of cysts, connective tissue
Cysts in cortex and medulla, seen with aneurysms and mitral valve prolapse
Defective PCT bicarbonate reuptake, hypokalemia, acidosis
Forms staghorn calculi, caused by urease positive bacteria
Malignancy of PCT cells, clear cytoplasm
Increased urine pH, acidosis, nonfunctional collecting tubule cells
Crescents, MPO-ANCA, no IF deposits
Wedge shaped crystals, caused by hypocitraturia
Eosinophils in urine, drug hypersensitivity reaction
Mesangial proliferation, IgA deposits on IF
Wire loop capillaries, caused by SLE
Common in kids, effaced podocytes, responds to steroids
Caused by PCT amino acid transporter defect, seen in children
Secondary hyperPTH due to decreased Vitamin D production
Found in children, contains embryonic tissue, found in Beckwith-Wiedemmann
Hematuria, arthralgias, rash, GI distress
Inherited defect in NaCl transporter in DCT
Linear IF deposits, crescents, anti-GBM antibodies
Segmental sclerosis of glomeruli, foot process effacement, common in AA/hispanics
Cortical infarction caused by DIC from obstetric issues
Upper/Lower respiratory symptoms, crescents, PR3-ANCA, no IF deposits
Inherited defect in Na/K/2Cl transporter
Cystic dilation of collecting ducts, can cause oligohydramnios
Envelope crystals, caused by increased urine pH
Tram track deposits, 2nd type due to C3 nephritic factor

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Created Mar 12, 2016ReportFavoriteNominate
Tags:pathology, usmle

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