Science Quiz / USMLE Renal Pathologies

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Can you name the USMLE Renal Pathologies?

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Caused by PCT amino acid transporter defect, seen in children
Wire loop capillaries, caused by SLE
Flank pain, eosinophilic casts, thyroidization of tubules
Upper/Lower respiratory symptoms, crescents, PR3-ANCA, no IF deposits
Common in kids, effaced podocytes, responds to steroids
Decreased renal blood flow causes tubular ischemia
Defective PCT bicarbonate reuptake, hypokalemia, acidosis
Crescents, MPO-ANCA, no IF deposits
Malignancy of PCT cells, clear cytoplasm
Sporadic defect, nonfunctional kidney made of cysts, connective tissue
Tubulointerstitial fibrosis, unable to concentrate urine, poor prognosis
Associated with gout, not seen on Xray
Mass with central scar, eosinophilic cells, no perinuclear clearing
Secondary hyperPTH due to decreased Vitamin D production
Increased urine pH, acidosis, nonfunctional collecting tubule cells
Hematuria, arthralgias, rash, GI distress
Hematuria, basket weave GBM, eye/ear pathologies
Hyperkalemia, decreased urine pH, hypoaldosteronism
Forms staghorn calculi, caused by urease positive bacteria
Wedge shaped crystals, caused by hypocitraturia
ureteric bifurcation or doubling, increased UTI risk
Congo red stain, apple green birefringence
Tram track deposits, 2nd type due to C3 nephritic factor
Painless hematuria, caused by chronic S. haematobium infection
Decreased renal blood flow, increased BUN/creatinine ratio
Envelope crystals, caused by increased urine pH
HintAnswer
Cysts in cortex and medulla, seen with aneurysms and mitral valve prolapse
Mesangial proliferation, IgA deposits on IF
Cystic dilation of collecting ducts, can cause oligohydramnios
Sloughing of necrotic papilla, caused by diabetes, sickle cell, NSAIDs
Toxic necrosis of tubular epithelial cells in PCT
Linear IF deposits, crescents, anti-GBM antibodies
Inherited defect in NaCl transporter in DCT
Found in pelvis, trapped by IMA
Cortical infarction caused by DIC from obstetric issues
eosinophilic nodular glomerulosclerosis (Kimmelstiel Wilson)
Inherited defect, constituitively activates ENaC channel
Bilateral outflow tract obstruction, increased FENa
Overactive bladder, leakage with urge to void immediately
Found in children, contains embryonic tissue, found in Beckwith-Wiedemmann
Eosinophils in urine, drug hypersensitivity reaction
Coca cola urine, subepithelial humps, hypercellular glomeruli
Flank pain, WBC casts, neutrophils in interstitium
Painless hematuria, caused by cyclophosphamide, phenacetin, aniline dyes
Outlet incompetence, leakage with increased abdominal pressure
Suprapubic pain, dysuria, positive urine nitrites
Oligohydramnios leading to lung hypoplasia
Inherited defect in Na/K/2Cl transporter
Incomplete bladder emptying, leakage with overfilling
Segmental sclerosis of glomeruli, foot process effacement, common in AA/hispanics
Global defect with PCT resorption
Diffuse GBM thickening, spike and dome pattern on EM

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