Science Quiz / Warwick 2018-19 Block 3 Drugs

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Can you name the Warwick 2018-19 Block 3 Drugs?

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Drug CategoryDrug
Dopaminergic (DA synthetic agonist that can cross BBB to replace lost DA; Parkinson's)
Antibiotic (CNS infection e.g. bacterial meningitis)
AED (blocks VGSC in inactivated state to ↓neuronal excitability; neuropathic pain)
General Anaesthetic (IH; invasive procedures)
Barbiturate (GABA-A receptor (b-subunit) co-agonist; epilepsy)
Strong Opioid (acts on opioid-R to close Ca2+ channels & ↑K+ outflow to ↓transmission; analgesic)
Anticholinesterase (inhibits ACh breakdown to ↑ACh in the cleft; mild-to-mod dementia)
Barbiturate (GABA-A receptor (b-subunit) co-agonist; epilepsy)
NeuroMuscular Blocker (non-depolarising; competitive with ACh in cleft to prevent depolarisation; pre-surgery)
MAO-B Inhibitor (prevents CNS breakdown of levodopa by MonoAmine Oxidase-B; Parkinson's)
Dopa-decarboxylase Inhibitor (prevents peripheral breakdown of levodopa by dopa-decarboxylase; Parkinson's)
Weak Opioid (acts on opioid-R to close Ca2+ channels & ↑K+ outflow to ↓transmission; analgesic)
NeuroMuscular Blocker (depolarising; non-competitive but prevent depolarisation by overloading cleft with ACh; pre-surgery)
Atypical Antidepressant (SNRI; ↑5HT & NA in cleft by inhibiting reuptake pump)
Muscle Relaxant (b-Blocker)
COX2-specific NSAID (inhibit COX-2 enzymes to ↓AA breakdown into PGs, which is req. for VGSC activation, therefore ↓pain transmission; analgesic)
Strong Opioid (acts on opioid-R to close Ca2+ channels & ↑K+ outflow to ↓transmission; analgesic)
COX2-specific NSAID (inhibit COX-2 enzymes to ↓AA breakdown into PGs, which is req. for VGSC activation, therefore ↓pain transmission; analgesic)
AED (for neuropathic pain; GABA mimetic, acts on Ca2+ channels; increases GABA transport)
Dopa-decarboxylase Inhibitor (prevents peripheral breakdown of levodopa by dopa-decarboxylase; Parkinson's)
Partial / Mixed Agonist Opioid (analgesic)
Local Anaesthetic (blocks VGSC to prevent depolarisation)
1st Generation Antipsychotic (inhibit D2-R to ↓DA in mesolimbic pathways; schizophrenia)
Dopaminergic (DA synthetic agonist that can cross BBB to replace lost DA; Parkinson's)
NSAID (inhibit COX enzymes to ↓AA breakdown into PGs, which is req. for VGSC activation, therefore ↓pain transmission; analgesic)
AED (blocks VGSC in inactivated state to ↓neuronal excitability; focal seizures)
Anticholinergic (dopaminergic effect by re-balancing dopamine with ACh; Parkinson's)
Weak Analgesic / Antipyretic (analgesic)
Used to reverse NMB block
Atypical Antidepressant (NDRI; ↑NA & DA in cleft by inhibiting reuptake pump)
Drug CategoryDrug
AED (for neuropathic pain; Sodium channel blocker)
Muscle Relaxant (Alpha 2 adrenergic agonist)
Atypical Antidepressant (NRI; ↑NA in cleft by inhibiting reuptake pump)
Antibiotic (CNS infection e.g. viral encephalitis)
Glutamate/NDMA Receptor Antagonist (inhibits glutamate-mediated cell death; mod-to-severe dementia)
Muscle Relaxant
NeuroMuscular Blocker (depolarising/anticholinesterase; non-competitive but prevent depolarisation by overloading cleft with ACh; pre-surgery)
AED (for neuropathic pain; GABA mimetic, acts on Ca2+ channels; increases GABA synthesis)
COMT Inhibitor (prevents CNS breakdown of levodopa by COMT; Parkinson's)
General Anaesthetic (IV; invasive procedures)
Muscle Relaxant (BDZ)
TriCyclic Antidepressants (blocks NA & 5HT reuptake pumps to ↑levels in the cleft for transmission)
NSAID (inhibit COX enzymes to ↓AA breakdown into PGs, which is req. for VGSC activation, therefore ↓pain transmission; analgesic)
Local Anaesthetic (blocks VGSC to prevent depolarisation)
Opioid Receptor Antagonist (prevents binding of opioids to opioid receptors; overdose)
General Anaesthetic (IV; invasive procedures)
Anticholinergic (dopaminergic effect by re-balancing dopamine with ACh; Parkinson's)
Atypical Antidepressant (adrenergic; increases presynaptic release)
Used for Opioid Addiction Management
Antibiotic (CNS infection e.g. listeriosis)
Atypical Antidepressant (melatonin agonist)
Muscle Relaxant
AED (blocks VDCC to ↓excitability; generalised absence seizures)
General Anaesthetic (IH; invasive procedures)
NeuroMuscular Blocker (non-depolarising; competitive with ACh in cleft to prevent depolarisation; pre-surgery)
Benzodiazepine (GABA-A receptor (y-subunit) co-agonist; epilepsy/dystonias give via rectum in status epilepticus)
Used for Opioid Addiction Management
MAO-B Inhibitor (prevents CNS breakdown of levodopa by MonoAmine Oxidase-B; Parkinson's)
NSAID (inhibit COX enzymes to ↓AA breakdown into PGs, which is req. for VGSC activation, therefore ↓pain transmission; analgesic)
MonoAmine Oxidase-A Inhibitors (↑ NA/5HT levels by preventing enzymatic breakdown; depression)
Drug CategoryDrug
Benzodiazepine (GABA-A receptor (y-subunit) co-agonist; epilepsy/dystonias, give via IV in status epilepticus)
Selective Serotonin Reuptake Inhibitors (↑5HT in cleft by inhibiting reuptake pump; depression & first-line for ALL anxiety disorders except PTSD)
Anticholinesterase (inhibits ACh breakdown to ↑ACh in the cleft; mild-to-mod dementia)
Local Anaesthetic (blocks VGSC to prevent depolarisation)
2nd Generation Antipsychotic (5HT/DA antagonism to ↓mesolimbic transmission; schizophrenia)
Used to reverse NMB block (encapsulates non-depolarising NMB for removal)
Anticholinergic (dopaminergic effect by re-balancing dopamine with ACh; Parkinson's)
Dopaminergic (DA synthetic agonist that can cross BBB to replace lost DA; Parkinson's)
Corticosteroid (CNS infection)
Opioid Receptor Antagonist (prevents binding of opioids to opioid receptors; overdose)
TriCyclic Antidepressants (blocks NA & 5HT reuptake pumps to ↑levels in the cleft for transmission)
1st Generation Antipsychotic (inhibit D2-R to ↓DA in mesolimbic pathways; schizophrenia)
NSAID (inhibit COX enzymes to ↓AA breakdown into PGs, which is req. for VGSC activation, therefore ↓pain transmission; analgesic)
Atypical Antidepressant (Partial 5HT agonist)
Anticholinesterase (inhibits ACh breakdown to ↑ACh in the cleft; mild-to-mod dementia)
General Anaesthetic (IH; invasive procedures)
Weak Opioid (acts on opioid-R to close Ca2+ channels & ↑K+ outflow to ↓transmission; analgesic)
Dopaminergic (DA precursor that can cross BBB to replace lost DA; Parkinson's)
Barbiturate (GABA-A receptor (b-subunit) co-agonist; epilepsy)
COMT Inhibitor (prevents CNS breakdown of levodopa by COMT; Parkinson's)
Mood Stabiliser (Bipolar Disorder)
AED (blocks VGSC in inactivated state to ↓neuronal excitability; generalised seizures)
Muscle Relaxant
2nd Generation Antipsychotic (5HT/DA antagonism to ↓mesolimbic transmission; schizophrenia)
Selective Serotonin Reuptake Inhibitors (↑5HT in cleft by inhibiting reuptake pump; depression)
2nd Generation Antipsychotic (5HT/DA antagonism to ↓mesolimbic transmission; schizophrenia)
Benzodiazepine Antagonist (reverses BDZ-induced sedation)
Benzodiazepine (GABA-A receptor (y-subunit) co-agonist; epilepsy/dystonias, give via buccal in status epilepticus)
Dopamine Depletor (blocks VMAT2 to ↓DA released into synaptic cleft; Huntington's)
Muscle Relaxant (BDZ - 'Z-drug')

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Created Feb 2, 2018ReportNominate
Tags:2017, block, category, drug, warwick

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