Science Quiz / Cell Bio Lecture 25

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Can you name the Cell Bio Lecture 25?

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used for monitoring the environment prior to committing to replication
condensation of chromosomes before mitosis
replication of DNA
specialized resting state for neurons and skeletal muscle
Is all DNA replicated? Is all DNA damage repaired?
Are all chromosomes properly attached to mitotic spindle?
Is environent favorable?
have no enzymatic activity and vary in concentration throughout cell cycle
enzymatic activity after cyclin binding, concentration steady but activation state changes
Cyclins are degraded by ____ targeted degradation
function as molecular brakes on cell cycle
escape from G1 checkpoint requires
can be a reversible or irreversible path out of cell cycle
promotes assembly of pre-replicative complex
Cdc6 increases in early
activation of ___ activates replication machinery
S-Cdk phosphorylating Cdc and causing dissociation from ORC prevents
chromosomal duplication results in
sister chromatids are held together by
activates Cdk inhibitor p21
binds G1-S Cdk and S-Cdk, arresting cell cycle in G1 until DNA is repaired
activation of M-Cdk is triggered by
hintanswer
proteins that help chromosomal condensation
composed of microtubules and microtubule associated proteins, separates chromatids into daughter cells
composed of actin and myosin, carries out cytoplasmic division
centrosomes are duplicated in
microtubules grow out from centrosomes in
spindle microtubules connect to chromosomes via the
each kinetochore binds __-__ microtubules
destroys securin and M-cyclin
securin normally binds ___ and inhibits it
when securin is degraded, separase destroys
chromosomes are pulled poleward in
poles are pushed and pulled apart in
nuclear pore proteins and nucelar lamins are dephosphorylated in
after telophase, ____ can occur
In plants, this guides cytokinesis instead of contractile ring
bind to cell surface receptors and stimulate cell division, usually from releasing G1/S cell cycle blockade
binds transcription regulators, brake for cell division at G1 and G1/S phases, phosphorylated after mitogen activation
performs proteolytic cascade that is irreversible
proteins that regulate caspases
proteins that release cytochrome c from mitochondria
activates caspases
sustaining proliferative signaling, evading growth suppressors, activating invasion and metastasis, enabling replicative immortality, inducing angiogenesis, resisting cell death ar

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