Science Quiz / Respiratory

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Can you name the Respiratory?

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how is sound produced
when does sound pitch change
drugs that can admin through nasal psg
def: ventilation
what does the epi and mucous membrane line
what size particles are removed from nasal psg
what particles can enter
is o2 or co2 lipid soluble
treatment of congestion
alpha agonist action
ex of alpha agonist
pharynx function
pharynx connections
larynx function
larynx connection
laryngitis symptoms
paranasal sinuses function
why sinus infections takes longer to treat with ABs
trachea location
trachea branches
where in resp system is there cartilage
where is there no cartilage
function of cilia movement
how many alveoli per lung
alveoli diameter
alveoli total SA
surgical trachea procedure
thickness of resp/alv membrane
characteristics of alv side of membrane
resp memb SA for diffusion
how well can o2 and co2 diffuse across resp memb
resp mem function
steps of pulm circulation starting with tissues
steps of systemic circulation starting with lungs
steps of bronchial circulation starting with aorta
where is the pleural memb located
pleural cavity of each lung location
pleural fluid location
intrapleural P def and value
diaphragm contracts, effect on thoracic cavity
ext intercostal muscles contract, effect on ribs and thoracic cavity
what causes increased thor cav volume
what innervation do skeletal muscles get
what causes skel muscle paralysis
where do adrenergic neurons NOT act
effect of EPI on bronchoconstriction
where do adrenergic neurons act DIRECTLY
which adrenergic neuron causes vasoconstriction
how does NE cause vasoconstriction
where does EPI act
action of beta-2 rec
where are alpha rec located
where are beta rec located
beta-2 agonists that treat bronchoconstriction
long acting beta-2 agonists
ultra long acting beta-2 agonist
how do beta-2 agonists cause bronchodilation
beta-2 agonist secondary action
other beta-2 agonist actions
what does cholinergic stimulation cause
how does ach cause bronchoconstriction
example of M rec blocker
where does most cholinergic innervation happen
which M rec are in airways
M2 rec actions
M3 rec actions
M rec blocker effects
NANC inhibitory neurons
VIP, NO actions
which is the strongest b.dilator
NANC excitatory neurons
SP, NKA actions
where is His made and released
why His released
His action
how does his cause bconstriction
what does His do to vasculature
antihistamines action
ex of antihistamines
where are leukotrienes made
why leukotrieens relased
leukotrienes action
treatment for leukotriene bconstriction
LT-synth inhib name
ex of cysLT1-rec blockers
what are prostaglandins
which prosts are bronchoconstrictors
where is SE made
SE actions
treatment against SE bconstriction
adenosine-rec action
treatment of adenosine-rec bconstriction
what is theophylline
ex of tachynins
tachynins action
all transmitters that cause bconstriction
all transmitters that cause bdilation
what does intrapulmonary P = alveolar P = P(pul) equal when not breathing
what does intrapleural P = P(ip) equal when not breathing
what does transpulmonary P = P(tp) equal when not breathing
Pneumothorax pressures
the direction of ventilation is determined by
Pressures at end of expiration
P's at start of inspiration
P's at end of inspiration
P's at start of expiration
what cause P(pul) to decrease at the start of inspiration
what cause P(pul) to increase at the start of expiration
what causes forced inspiration
what causes forced expiration
whats the range of P(pul) in forced breathing
what's the rule of P(ip) relation to P(atm)
whats the range of P(ip) in forced breathing
def: compliance
decreased compliance means what for the force to fill the lungs
normal compliance
affect of emphysema on compliance
affect of lung surfactant on compliance
affect of arthritis on compliance
formula: pulmonary airflow =
what contributes to airflow resistance
why does upper bronchoconstriction not contrib to ressitance
what are the 4 lung volumes
def: tidal volume
def: insp reserve vol
def: exp reserve vol
def: residual vol
how is respiratory volume measured
what are the 4 lung capacities
total lung cap (a number)
vital cap (a number)
inspiratory cap (number)
functional residual cap
def: anatomical dead space
def: phys dead space
in healthy person, what's the relationship btwn anat and phys dead space
normal respiratory rate
def: min resp volume
formula: MRV =
normal MRV (Number)
def: alveolar ventilation
formula: alv vent =
AV per min (number)
whats the effect of surface tension on alveolar size
how does pulm surfactant help alveoli
what happens if surface tension increases to much
why does surfactant effect increase during expiration
when is surfactant made in fetuses
what does surfactant need to be made
whats the half life of surfactant
def: resp distress syndrome
what does a spirometer measure
def: forced vital capacity
def: forced expir volume
healthy relationship btwn FVC and FEV
what happens to FVC and FEV in restrictive disease (fibrosis)
what happens to FVC and FEV in obstructive disease (asthma)
def: peak expir flow
def: max exp flow rate
is MEFR effort-dependent
how to measure PEF
composition of air from greatest
def: Daltons law of partial p
def: Henrys law of gas solub in liquid
temp effect on gas sol in liquid
what type of transport is the exchange between alv cap blood & alv air
what type of transport is the exchange between tissue caps and cells
difference in H2O solubility of co2 and o2
whats the diff in pO2 of periph tissues and alveoli
what happens to interstitial & intracellular pO2 during exercise
what happens to pO2 and Hb saturation as altitude increases
whats the Hb saturation of pulmonary art and veins
whats the Hb saturation of systemic art and veins
how many heme in each Hb
how many Fe++ per heme
how are Hb saturation and pO2 related
what happens to oxy-hb when pO2 decreases
what factors affect oxygen binding to hb
decrease pH --> hb effect =
increase pCO2 --> hb effect =
increase temp --> hb effect =
increase DPG --> hb effect =
what causes hb deoxygenation
when does CO combine with Hb
how does CO combine with Hb
what are the 3 ways CO2 can be transported in blood
what are the 3 ways in order of most, to least, common
what are carbamino bonds
which form of Hb forms carbamino compounds better
do tissues or lungs have more carbamino compounds
how is bicarbonate formed
what enzyme forms carbonic acid
why does chloride shift occur
what are medulla inspiratory neurons called
what does the pons' pneumotaxic area do
what does the pons' apneustic area do
what are medulla excitatory neurons called
when are VRG neurons active
what do lung stretch rec do
when does pCO2 cause increased alv ventilation
when does pH cause increased alv ventilation
peripheral chemorec affected by
what does exercise do o2 consumption, Cardiac output, MRV
what do CNS stimulates do to resp activity
what happens with age to compliance, MRV, ventilation
what happens in emphesyma

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Created Apr 9, 2012ReportNominate

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