inhibits reabsorption of Na in CD by inhibiting production of renin, angII, and release of aldosterone, also causes dialation of aff arteioles to increase GFR

constricts aff arterioles and promotes Na reabsorption via Na/H antiporter

promotes angII, levels regulated by SYMP and amount of NaCl delivered to macula densa

increases SVR and BP, constricts aff and eff arterioles, increases Na reabsorption from PCT via Na/H antiporter and in CD via ENaC, increases aldosterone secretion, stimulates thir

increases Na reabsorption by DCT and CD and stimulates secretion of K and H via protein synthesis and stimulates release of AngII

increases water reabsorption by phosphorylating AQP2 channels and inserting them in the apical membrane

maintains constant GFR at differing arterial pressures via myogenic response to tubuloglomerular feedback

Beta AR nonspecific, used for treating HTN, should NOT be used in patients with asthma

Beta AR nonspecific, has intrinsic sympathomimetic activity, is a partial agonist

muscarinic R blocking agent, causes cycloplegia, mydriasis, decreased secretions/motility in GI tract

reversible AchE inhibitor, used for non-obstructive bladder/GI atony and myasthenia gravis

neuromuscular blocking drugs, cause skeletal muscle paralysis

irreversible AchE inhibitor, can cause death

frees AchE from inhibition

muscarinic receptor agonist, causes vasodilation, bradycardia, hypotension,bronchoconstriction can exacerbate peptic ulcers, cause SLUDS

3rd generation alpha/beta nonspecific, good for long-term treatment of CHF, HTN or pheochromocytoma