| Theory | Researcher | Year |
| training causes lower total cholesterol, LDL cholesterol and circulating TG and higher HDL cholesterol | |
| obesity = decreased adiponectin = decreased AMPK = decreased fat oxidation in muscles | |
| exercising 12 hours before a high fat meal gives the greatest benefit to lipaemic respone | |
| TNF alpha causes decreased GLUT4 translocation | |
| increased insulin causes vasconstriction in the absence of NO | |
| training decreases blood pressure via decreased sympathetic nerve activity | |
| increased exercise intensity = decreased clotting time | |
| training increases LPL gene expression | |
| ghrelin is switched off by insulin | |
| high apoB-apoA ratio increases the thickness of vessel walls and causes atherosclerosis | |
| ERSCD in old caused by plaque rupture... plaque rupture caused by increased macroghage and lipid content of plaque and decreased smooth muscle content | |
| ApoA makes LDL resistant to oxidation | |
| increased intensity = increased platelet count | |
| HDL inhibits adhesion molecules | |
| exercise decreases glycogen which increases IL6 in muscles. IL6 increases IL10 which increases the TH2 response | |
| acute exercise decreases plassminogen activator inhibitor | |
| training decreases apoB | |
| obesity and inactivity are not decreasing | |
| increased walking speed = decreased coronary heart disease | |
| 50% increase in statin use from 1997 to 2002 | |
| fitness is more important than activity when battling cardiovascular disease | |
| high cholesterol, high fasting glucose, smoking, low fitness and hypertension increase mortality | |
| people sit too much and should do more non-exercise physical activity | |
| obestiy = increased fatty acid metabolites in muscle = increased PKC theta = increased serine phosphorylation of IRS-1 | |