Exam 7 Pharmacology Part 2

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Can you name the Drugs for Exam 7 Pharmacology Part 2?

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Molecular ActionDrugDisease or Use
α1β antagonist, reduces inotropy and chronotropy, vasodilation, anti-oxidant, anti-apoptotic activityHypertension (esp. with hyperlipidemia or diabetes), systolic congestive heart failure, diastolic congestive heart failure, stable angina
Inhibits angiotensin-converting enzyme (ACE), arterial vasodilation, reverses cardiac and arterial remodeling, reduced aldosterone secretionHypertension (primary drug in cases of diabetes, post-MI, and chronic renal disease), post-MI prophylaxis against the development of congestive heart failure, systolic and diastolic congestive heart failure
Blocks vascular L-type calcium channels, stabilizes inactivated state of those calcium channels, reduces myocardial oxygen demand, arterial vasodilationHypertension (usually as third drug or as second drug in isolated systolic hypertension), stable angina (as adjunct to nitrate + beta blocker), variant angina
(2) Block potassium channels, varying effects on sodium and calcium channels, inhibit repolarization in the atria and ventriclesPrevent cardiac tachyarrythmias, terminate acute ventricular cardiac tachyarrythmias
Block potassium channels, inhibit repolarization in the atria and ventriclesPrevent cardiac tachyarrhythmias
α1 receptor antagonist, vasodilationSecond-line hypertension (esp. in persons with high LDL and low HDL)
Inhibits Na/K ATPase --> greater delivery of Ca to the contractile apparatus of cardiomyocytes, inc. activity in the vagus nerve, slows conduction, inc. refractoriness in AV nodeSystolic congestive heart failure, supraventricular arrythmias
(2) Block cardiac and vascular L-type calcium channels, stabilize inactivated state of those calcium channels, arterial vasodilation, reduce inotropy and chronotropy, reduce myocarStable angina (as alternative to beta blocker esp. in persons with persistent asthma), diastolic congestive heart failure (as alternative to beta blocker esp. in persons with persistent asthma), supraventricular tachyarrhythmias
β1 antagonist/increases NO availability, reduces inotropy and chronotropy, vasodilation, anti-remodeling activityHypertension (esp. in persons who experience exercise intolerance or sexual dysfunction when treated with another beta blocker), diastolic congestive heart failure, stable angina
(name both: _ + _) Source of NO (1st) + inhibitor of ROS production (2nd), dilation of veins more than arteries, anti-remodeling activityCongestive heart failure (African-Americans)
Molecular ActionDrugDisease or Use
AT1 receptor antagonist (ARB), otherwise same as LisinoprilHypertension (primary drug in cases of diabetes, post-MI, and chronic renal disease), post-MI prophylaxis against the development of congestive heart failure, systolic and diastolic congestive heart failure
Blocks the pacemaker channel in the SA node (HCN4), reduces heart rate without affecting inotropy or blood pressure, reduces myocardial oxygen demandStable angina
(2) Sources of NO in vascular smooth muscle, dilation of veins more than arteries (Reduce myocardial O2 demand by red. preload via venous dilation)Stable and unstable angina, acute decompensated congestive heart failure (patch)
Blocks sodium channels, stabilizes inactivated state of sodium channels, slows conduction in the ventricles, increases the effective refractory period in the ventriclesTerminates acute ventricular tachyarrhythmias
Activates A1 receptors in AV node, slows conduction in the AV node, increases the effective refractory period in the AV nodeTerminates acute supraventricular tachyarrythmias
Increases cGMP in vascular smooth muscle, suppresses renin-angiotensin systemAcute decompensated congestive heart failure
(2) β1-selective antagonists, reduce inotropy and chronotropy, *reduce myocardial oxygen demand*, slow conduction and increase refractoriness in AV node, reverse cardiac and arterHypertension ( although being downgraded for this use), stable and unstable angina, diastolic congestive heart failure, supraventricular tachyarrythmias
Non-selective β receptor antagonist, blocks potassium channels, slows conduction in the AV node, increases the effective refractory period in the AV node, inhibits repolarization Prevents cardiac tachyarrhythmias
Blocks late sodium channel, reduces inotropy without affecting heart rate or blood pressure, reduces myocardial oxygen demandStable angina

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