Can you name the heart & circulation drug? created by klamberry Enter a drug in the box below Correctly named drugs will show up below Answers do not have to be guessed in order This quiz has not been verified by Sporcle

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Atrial Fibrillation anti-thrombotic   anti-thrombotic if additional risk factors   slows AV node, minor vagal response, slow onset (side fx: heart block, any arrhythmia)   slows AV node and makes it more refractory (class 4, side fx: negative inotrope, X ind.: beta blocker)   slows AV node and makes it more refractory (class 4, side fx: negative inotrope, X ind.: beta blocker)   slows AV node by reducing sympathetic stimulation   electric solution for AF   restores sinus rhythm and slows AV node, prophylactic (multiblock, side fx: variable, hypotension, bradycardia, skin, pulmonary, and thyroid toxicity)   restores sinus rhythm and slows AV node (inward K block, nonselective beta block, side fx: long QT)   *restores sinus rhythm and slows AV node (class IC, side fx: conduction block, bradycardia)   *restores sinus rhythm and slows AV node, patients with good ventricle (multiblock, side fx: less than other multiblock)	SVT temporary AV node block, diagnostic (X indic.: asthma)   slows AV node and makes it more refractory (class 4, side fx: negative inotrope)   slows AV node and makes it more refractory (class 4, side fx: negative inotrope)   chronic 1st choice: slows AV node by reducing sympathetic stimulation   surgical solution   restores sinus rhythm and slows AV node, prophylactic (multiblock, side fx: variable, hypotension, bradycardia, skin, pulmonary, and thyroid toxicity)   restores sinus rhythm and slows AV node (class 1a, side fx: pro-arrhythmia, anticholinergic)   restores sinus rhythm (class 3, side fx: long QT)	VT temporary AV node block, diagnostic (X indic.: asthma)   acute, monomorphic ischemic, decreases automaticity (class 1b, hepatic metabolism, side fx: seizures, CV collapse)   acute, any monomorphic, prolongs action potential (class 1a, side fx: pro-arrhythmia, anticholinergic)   acute or chronic, slows automaticity (multiblock, side fx: variable, hypotension, bradycardia, skin, pulmonary, and thyroid toxicity)   chronic, lengthens action potential, decreases automaticity (inward K block, nonselective beta block, side fx: long QT)   *chronic, reduced conductivity in ventricle (class IC, side fx: conduction block, bradycardia)   electric solution
Sympathetic response sympathomimetic, resuscitation, counters anaphylaxis, local vasoconstriction, released by adrenal medulla   released by SNS post-ganglionic neuron for heart   doesn't cross BBB, increases blood pressure and cardiac output   acute treatment of bradycardia, elevates heart rate and vasodilates   causes intense vasoconstriction, used as a decongestant and vasopressor   mixed alpha/beta effect, indirect release of NE to cause SNS symptoms, used as a stimulant, decongestant, and appetite suppressant   bronchodilator, may cause heart palpitations and tremor   anti-muscarinic, used in cardiac resuscitation, hot as a hair, blind as a bat, DRY AS A BONE, red as a beet, mad as a hatter   inhaled as a bronchodilator, anti-muscarinic	Parasympathetic response vasodilation, treatment for pheochromocytoma   antihypertensive, causes vasoconstriction with little effect on heart   used to treat glaucoma, myasthenia gravis and to reverse neuromuscular blockers   quick acting, causes muscle fasciculations then paralysis, unreversible by AchEI   causes paralysis, reversible by AchEI   complicated response, SNS and PSNS depending on organ tone and dose (blockade at high dose)   treats hypertension but as a last resort, decreases SNS   anti-hypertensive, reduces heart contractility and vasodilates	Hypertension reduces vasoconstriction, increases Na/H2O secretion to drop BP (side fx: angioedema, cough, hyperkalemia, worse kidney)   reduces vasoconstriction, increases Na/H2O secretion to drop BP (side fx: hyperkalemia)   causes vasodilation to reduce afterload, some reduce heart contractility as well (side fx: bradycardia, hypotension)   blocks sympathetic response preventing elevated contractility, causes vasoconstriction   increases H2O excretion to reduce blood volume and BP (side fx: maybe hyperkalemia)   healthy diet, exercise   restrict EtOH, restrict Na intake, smoking cessation, control dyslipidemia, CPAP
Cardiac failure to reduce fluid overload/pulmonary edema (side fx: hypokalemia)   1st line reduces vasoconstriction and afterload, increases Na/H2O secretion to drop BP (side fx: angioedema, cough, hyperkalemia, worse kidney)   1st line reduce chronic sympathetic stimulation on heart   2nd line increased contractility, reduces hospital admissions but not mortality (side fx: arrhythmia, increased AV block, yellow-green halo)   2nd line increases H2O and Na secretion (side fx: hyperkalemia)   only if other drugs fail, vasodilates to reduce afterload   electrical solution, provides a shock if heart enters arrhythmia   if a suitable candidate, extends lifespan by 10-15 years   exercise   smoking cessation, reduce Na consumption, control DM, fluid restriction	Dyslipidemia anti-HMG-CoA causing upregulation of LDLr and cholesterol excretion, LDL-C down by ~33% (side fx: CK elevated, myalgia, rhabdomyolysis, elevated LFT)   raises HDL-C, inhibits VLDL secretion, activates LPL for VLDL clearance   multiple sites of action, reduces LDL-C, better at lowering TG (side fx: potentiates warfarin, elevated LFT)   safe alternative for reducing TG   blocks cholesterol re-uptake from intestine, LDL-C down by ~15%   blocks cholesterol uptake by sequestration   antioxidant for treatment of homozygous familial hypercholesterolemia	Coronary artery disease if coronary angioplasty is unavailable, to remove plaque   common anti-platelet, provided upon MI admission   anti-platelet if other is contraindicated   anti-coagulant administered upon MI admission   control dyslipidemia   control hypertension   vasodilation for angina relief prn, provided upon MI admission
Bradycardia acute treatment, stimulates SNS response   acute treatment, anti-muscarinic/anti-PSNS response   acute treatment, beta agonist stimulates heart rate   chronic treatment

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