| Hints | |
|---|
| slowly-developing (3-6 years) 'lumpy' or discolored lesions lead to sensory nerve damage and tissue | |
| toxin causes complications; respiratory failure causes death in 70% of patients | |
| skin disfiguration from chronic benign tumors | |
| infection leads to sudden malaise, fever, muscle pain, headache and sore throat followed by vomiting, diarrhea, rash and heavy bleeding in 2-21 days; complications include massive | |
| mosquito 'injects' virus into bloodstream; virus replicates in the CNS, causing fever and headache, vomiting, stiff back and neck, drowsiness, tremors, loss of coordination, convul | |
| rapidly-spreading infection leads to destruction of tissues at sites of infection ... aka Flesh-Eating Disease | |
| grows on the skin, it induces a bright red inflammation, blister-like superficial lesions or more severe lesions all of which are perhaps related to hypersensitivity | |
| mosquito 'injects' virus into bloodstream where the virus replicates in the liver (causing jaundice), intestines (causing bloody stools and, sometimes, vomiting of blood), and gin | |
| mosquito 'injects' virus into bloodstream; this rarely fatal disease is characterized by sudden onset of high fever and rash together with severe headache, and severe muscle, join | |
| infection of liver and erythrocytes causes damage leading to chronic recurrent chills followed by high fever (104 F causes delirium, convulsions), sweating, headache, nausea, sple | |
| damage to blood vessels causes fever headache, muscle pains; complications - damage to blood vessels throughout the body leads to impaired function of the kidneys, liver, lungs, C | |
| overproduction of sebum causing skin to swell over pore | |
| | Hints | |
|---|
| 60-85% of patients develop an expanding red rash (erythema chronicum migrans or ECM) at the site of a tick-bite, ring-shaped after 3 days to one month; accompanied by flu-like sym | |
| superficial infection leads to cellulitis, then to myonecrosis due to toxins; complications - death due to CNS damage caused by toxins (sometimes complicated by gas formation, henc | |
| skin lesions (painless pink, red or purplish 'bumps') develop at site of infection after 3-12 weeks, develop into ulcers that don't heal, then enter the lymphatics and 'move' up ar | |
| cercariae burrow through skin, then infect the bloodstream (causing fever, chills, sweats; back, groin or leg pain; urticaria), then infect the liver, where mature flukes develop a | |
| infects and kills macrophages; whole body (liver, spleen, lymph nodes (buboes), lungs, meninges, skin) damaged, causing fever and massive blood clotting, which leads to shock, whi | |
| recurrent chronic lesions (blisters) on face, fingers, hands ... becomes latent in local nerves and exhibits recurrence during periods of stress (physical or psychological); herpe | |
| superficial infection leads to formation of thin-walled abscesses that look like blisters or 'weeping' lesions with scabs | |
| tsetse fly bite becomes painful and swollen in several days, then a lesion like a syphilitic chancre develops at site; then systemic infection via bloodstream develops, causing fe | |
| inflammation of single hair follicle or sebaceous gland that swells with pus | |
| superficial skin infection causes granulomas on head/scalp | |
| as endospores germinate, the vegetative bacterial cells begin to produce a necrotizing toxin that causes development of pustular lesions called eschars characterized by necrotic ce | |
| encephalitide that causes neurological complications that lead to twitching paralyzation, disorientation and hyperactivity | |
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Multiplication Table
Minute Math (Addition)
