| Question | Answer |
|---|
| Where is somatostatin produced in the hypothalamus? | |
| Main target organ for growth hormone that causes its long term effect related to growth | |
| Insulin and IGF-1 receptors are apart of what class of receptors? | |
| T/F: The chronic release of GH causes an increase in plasma glucose and increase in plasma insulin, leading to hyperglycemia and insulin resistance. | |
| T/F: GH acts on proteins by increasing the amino acid intake, increasing protein synthesis, increasing transcription, and decreasing protein degradation. | |
| This receptor is a clearance receptor that removes its ligand out of circulation and into the cell to undergo lysosomal degradation? | |
| T/F: pvGH is produced by the placenta. It binds to hGH receptor and is extremely important for the development of the fetus. | |
| T/F: Levels of GH increase significantly during the day. | |
| obesity ______ the release of GH | |
| T/F: When GHRH binds to its receptor intracellularly, it increases cAMP levels and causes an influx of calcium and release of secretory vesicles containing GH. | |
| glucagon acts to _______ the release of GH. | |
| this condition is caused by excessive GH before adolescence, which causes increased body size and hyperglycemia. | |
| T/F: Growth hormone acts to increase glucose uptake. | |
| T/F: Somatostatin can only be made in the hypothalamus. | |
| this pathological condition is caused by a deficiency of GH or IGF-1 during childhood. Patients are short (but proportional) with no mental retardation. | |
Digits of Pi
Multiplication Table
Minute Math (Addition)
