Gastric Physiology

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Can you name the Gastric Physiology?

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T/F: Proton pump inhibitors bind irreversibly to the H/K proton pump.
These cells in the stomach are activated by the H+ions in the stomach to reduce the release of gastrin
this hormone acts on the G cell in the antrum of the stomach to inhibit the release of gastrin
occurs due to a defiency in vitamin B12, which inhibits the body's ability to go through erythropoesis
H2 blockers prevent the binding of ____ to prevent the production of acid.
T/F: Ileal fat increase gastric emptying.
occurs due to uncontrolled gastric emptying due to the lack of feedback inhibition by the duodenum and undigested food makes it to the colon
these cells produce somatostatin
T/F: Prilosec or Omeprazole acts at the basolateral membrane of the parietal cell.
This type of drug inhibits prostaglandin related defense mechanisms within the stomach
T/F: An increase in pH (basicity) slows gastric emptying.
T/F: The stomach is the major site for digestion.
these cells produce pepsinogen and gastric lipase
what activates pepsinogen?
T/F: Tagemet and cimetidine prevent acid production by blocking H1 histamine receptors
Cl ions are transported into the lumen of the stomach using which type of transport protein?
P/D1 cells are located where?
T/F: Rate of gastric emptying increases with rectal and colonic distention.
these cells produce gastrin
these cells produce histamine
T/F: ECL cells can be activated by acetylcholine or gastrin
failure of the pyloric sphincter to relax for gastric emptying... leads to projectile vomiting
pespinogen and HCl are released into the lumen of the stomach... where is histamine released?
The superficial epithelial cells along with which other cell type in the stomach act to lubricate and protect the stomach wall?
___ are hormones released by the duodenum and cause feedback on stomach to decrease gastric emptying and secretions
Hypoglycemia ___ (increase or decreases) gastric emptying.
The movement of Cl into the parietal cell is coupled to the movement of which ion out of the cell?
severely delayed gastric emptying or no emptying
QuestionAnswer
2 causes of peptic ulcers
T/F: Ghelin is released during fasting which stimulates appetite
which two products of oxidative phosphorylation are important for the production of stomach acid.
made by the S cells of the duodenum
How does potassium get out of the parietal cell?
these cells produce HCl and intrinsic factor
CCK ____ (promotes or inhibits) gastric emptying
2nd messenger involved in activation of the parietal cell
The gland of the stomach is known an ____ gland.
responsible for getting H+ ions into the lumen of the stomach
Which histamine receptor is found on the basolateral membrane of the parietal cell?
prilosec or omeprazole acts by inhibiting what?
T/F: The parietal cell can be activated by Acetylcholine, histamine, or gastrin.
Secretin ____ (promotes or inhibits) gastric emptying
P/D1 cells release what hormone?
Nutrients in the ileum and the colon influence gastric emptying through the _____ mechanism
enzyme important for the production of protons and bicarbonate in the parietal cell
made by the I cells of the duodenum
Cl ions are transported into the parietal cell using which type of transport protein?
T/F: Large volumes in the stomach increase gastric emptying
leptin is release by which type of tissue?
essential for binding and absorption of vitamin B12
this hormone acts at the hypothalamus to suppress appetite
T/F: In some cases of peptic ulcer disease, acid from the stomach erodes the D cells of the stomach preventing the brake mechanism for acid production.
T/F: After a large meal, the alkaline tide (removal of bicarbonate from the parietal cell) causes an increase in the pH of the blood.
Peptide tyrosine-tyrosine (PYY) and glucagon like peptide (GLP1 and GLP2) ____ (stimulates/inhibits) gastric emptying
All stomach cell types are activated by the neurotransmitter (from the enteric nervous system) Acetylcholine, except which cell?

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Created Mar 25, 2012ReportNominate
Tags:physiology