Gastric Physiology

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QuestionAnswer
2nd messenger involved in activation of the parietal cell
H2 blockers prevent the binding of ____ to prevent the production of acid.
enzyme important for the production of protons and bicarbonate in the parietal cell
T/F: After a large meal, the alkaline tide (removal of bicarbonate from the parietal cell) causes an increase in the pH of the blood.
T/F: Ileal fat increase gastric emptying.
these cells produce gastrin
T/F: Tagemet and cimetidine prevent acid production by blocking H1 histamine receptors
what activates pepsinogen?
T/F: ECL cells can be activated by acetylcholine or gastrin
Which histamine receptor is found on the basolateral membrane of the parietal cell?
These cells in the stomach are activated by the H+ions in the stomach to reduce the release of gastrin
made by the S cells of the duodenum
The superficial epithelial cells along with which other cell type in the stomach act to lubricate and protect the stomach wall?
this hormone acts at the hypothalamus to suppress appetite
2 causes of peptic ulcers
prilosec or omeprazole acts by inhibiting what?
This type of drug inhibits prostaglandin related defense mechanisms within the stomach
this hormone acts on the G cell in the antrum of the stomach to inhibit the release of gastrin
leptin is release by which type of tissue?
How does potassium get out of the parietal cell?
severely delayed gastric emptying or no emptying
Hypoglycemia ___ (increase or decreases) gastric emptying.
CCK ____ (promotes or inhibits) gastric emptying
T/F: An increase in pH (basicity) slows gastric emptying.
occurs due to uncontrolled gastric emptying due to the lack of feedback inhibition by the duodenum and undigested food makes it to the colon
T/F: The parietal cell can be activated by Acetylcholine, histamine, or gastrin.
failure of the pyloric sphincter to relax for gastric emptying... leads to projectile vomiting
P/D1 cells are located where?
QuestionAnswer
The gland of the stomach is known an ____ gland.
T/F: In some cases of peptic ulcer disease, acid from the stomach erodes the D cells of the stomach preventing the brake mechanism for acid production.
The movement of Cl into the parietal cell is coupled to the movement of which ion out of the cell?
Cl ions are transported into the parietal cell using which type of transport protein?
T/F: Prilosec or Omeprazole acts at the basolateral membrane of the parietal cell.
these cells produce somatostatin
responsible for getting H+ ions into the lumen of the stomach
these cells produce HCl and intrinsic factor
Peptide tyrosine-tyrosine (PYY) and glucagon like peptide (GLP1 and GLP2) ____ (stimulates/inhibits) gastric emptying
P/D1 cells release what hormone?
T/F: Rate of gastric emptying increases with rectal and colonic distention.
which two products of oxidative phosphorylation are important for the production of stomach acid.
All stomach cell types are activated by the neurotransmitter (from the enteric nervous system) Acetylcholine, except which cell?
these cells produce histamine
pespinogen and HCl are released into the lumen of the stomach... where is histamine released?
Cl ions are transported into the lumen of the stomach using which type of transport protein?
Secretin ____ (promotes or inhibits) gastric emptying
T/F: Ghelin is released during fasting which stimulates appetite
made by the I cells of the duodenum
___ are hormones released by the duodenum and cause feedback on stomach to decrease gastric emptying and secretions
occurs due to a defiency in vitamin B12, which inhibits the body's ability to go through erythropoesis
essential for binding and absorption of vitamin B12
T/F: Proton pump inhibitors bind irreversibly to the H/K proton pump.
these cells produce pepsinogen and gastric lipase
Nutrients in the ileum and the colon influence gastric emptying through the _____ mechanism
T/F: Large volumes in the stomach increase gastric emptying
T/F: The stomach is the major site for digestion.

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