Gastric Physiology

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Can you name the Gastric Physiology?

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T/F: Rate of gastric emptying increases with rectal and colonic distention.
All stomach cell types are activated by the neurotransmitter (from the enteric nervous system) Acetylcholine, except which cell?
T/F: An increase in pH (basicity) slows gastric emptying.
The movement of Cl into the parietal cell is coupled to the movement of which ion out of the cell?
2nd messenger involved in activation of the parietal cell
this hormone acts on the G cell in the antrum of the stomach to inhibit the release of gastrin
severely delayed gastric emptying or no emptying
made by the S cells of the duodenum
T/F: The parietal cell can be activated by Acetylcholine, histamine, or gastrin.
these cells produce HCl and intrinsic factor
what activates pepsinogen?
these cells produce gastrin
Which histamine receptor is found on the basolateral membrane of the parietal cell?
pespinogen and HCl are released into the lumen of the stomach... where is histamine released?
occurs due to uncontrolled gastric emptying due to the lack of feedback inhibition by the duodenum and undigested food makes it to the colon
T/F: Prilosec or Omeprazole acts at the basolateral membrane of the parietal cell.
T/F: Proton pump inhibitors bind irreversibly to the H/K proton pump.
T/F: Large volumes in the stomach increase gastric emptying
these cells produce histamine
This type of drug inhibits prostaglandin related defense mechanisms within the stomach
2 causes of peptic ulcers
essential for binding and absorption of vitamin B12
___ are hormones released by the duodenum and cause feedback on stomach to decrease gastric emptying and secretions
enzyme important for the production of protons and bicarbonate in the parietal cell
this hormone acts at the hypothalamus to suppress appetite
Secretin ____ (promotes or inhibits) gastric emptying
Hypoglycemia ___ (increase or decreases) gastric emptying.
Cl ions are transported into the parietal cell using which type of transport protein?
responsible for getting H+ ions into the lumen of the stomach
P/D1 cells are located where?
P/D1 cells release what hormone?
T/F: After a large meal, the alkaline tide (removal of bicarbonate from the parietal cell) causes an increase in the pH of the blood.
occurs due to a defiency in vitamin B12, which inhibits the body's ability to go through erythropoesis
these cells produce pepsinogen and gastric lipase
T/F: The stomach is the major site for digestion.
T/F: ECL cells can be activated by acetylcholine or gastrin
The superficial epithelial cells along with which other cell type in the stomach act to lubricate and protect the stomach wall?
The gland of the stomach is known an ____ gland.
Nutrients in the ileum and the colon influence gastric emptying through the _____ mechanism
H2 blockers prevent the binding of ____ to prevent the production of acid.
Peptide tyrosine-tyrosine (PYY) and glucagon like peptide (GLP1 and GLP2) ____ (stimulates/inhibits) gastric emptying
CCK ____ (promotes or inhibits) gastric emptying
prilosec or omeprazole acts by inhibiting what?
which two products of oxidative phosphorylation are important for the production of stomach acid.
leptin is release by which type of tissue?
How does potassium get out of the parietal cell?
T/F: Ghelin is released during fasting which stimulates appetite
T/F: Tagemet and cimetidine prevent acid production by blocking H1 histamine receptors
Cl ions are transported into the lumen of the stomach using which type of transport protein?
T/F: Ileal fat increase gastric emptying.
made by the I cells of the duodenum
T/F: In some cases of peptic ulcer disease, acid from the stomach erodes the D cells of the stomach preventing the brake mechanism for acid production.
These cells in the stomach are activated by the H+ions in the stomach to reduce the release of gastrin
failure of the pyloric sphincter to relax for gastric emptying... leads to projectile vomiting
these cells produce somatostatin

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Created Mar 25, 2012ReportNominate