Gastric Physiology

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Can you name the Gastric Physiology?

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Hypoglycemia ___ (increase or decreases) gastric emptying.
2nd messenger involved in activation of the parietal cell
these cells produce pepsinogen and gastric lipase
H2 blockers prevent the binding of ____ to prevent the production of acid.
what activates pepsinogen?
The movement of Cl into the parietal cell is coupled to the movement of which ion out of the cell?
The gland of the stomach is known an ____ gland.
occurs due to uncontrolled gastric emptying due to the lack of feedback inhibition by the duodenum and undigested food makes it to the colon
severely delayed gastric emptying or no emptying
these cells produce somatostatin
responsible for getting H+ ions into the lumen of the stomach
pespinogen and HCl are released into the lumen of the stomach... where is histamine released?
T/F: Ileal fat increase gastric emptying.
2 causes of peptic ulcers
T/F: Rate of gastric emptying increases with rectal and colonic distention.
essential for binding and absorption of vitamin B12
T/F: The stomach is the major site for digestion.
failure of the pyloric sphincter to relax for gastric emptying... leads to projectile vomiting
enzyme important for the production of protons and bicarbonate in the parietal cell
T/F: Prilosec or Omeprazole acts at the basolateral membrane of the parietal cell.
this hormone acts on the G cell in the antrum of the stomach to inhibit the release of gastrin
P/D1 cells release what hormone?
These cells in the stomach are activated by the H+ions in the stomach to reduce the release of gastrin
T/F: After a large meal, the alkaline tide (removal of bicarbonate from the parietal cell) causes an increase in the pH of the blood.
this hormone acts at the hypothalamus to suppress appetite
made by the S cells of the duodenum
these cells produce histamine
these cells produce gastrin
P/D1 cells are located where?
Nutrients in the ileum and the colon influence gastric emptying through the _____ mechanism
occurs due to a defiency in vitamin B12, which inhibits the body's ability to go through erythropoesis
Secretin ____ (promotes or inhibits) gastric emptying
T/F: The parietal cell can be activated by Acetylcholine, histamine, or gastrin.
leptin is release by which type of tissue?
T/F: Proton pump inhibitors bind irreversibly to the H/K proton pump.
which two products of oxidative phosphorylation are important for the production of stomach acid.
All stomach cell types are activated by the neurotransmitter (from the enteric nervous system) Acetylcholine, except which cell?
How does potassium get out of the parietal cell?
This type of drug inhibits prostaglandin related defense mechanisms within the stomach
The superficial epithelial cells along with which other cell type in the stomach act to lubricate and protect the stomach wall?
these cells produce HCl and intrinsic factor
T/F: ECL cells can be activated by acetylcholine or gastrin
Cl ions are transported into the parietal cell using which type of transport protein?
Cl ions are transported into the lumen of the stomach using which type of transport protein?
T/F: Large volumes in the stomach increase gastric emptying
made by the I cells of the duodenum
T/F: An increase in pH (basicity) slows gastric emptying.
T/F: Ghelin is released during fasting which stimulates appetite
T/F: Tagemet and cimetidine prevent acid production by blocking H1 histamine receptors
___ are hormones released by the duodenum and cause feedback on stomach to decrease gastric emptying and secretions
prilosec or omeprazole acts by inhibiting what?
CCK ____ (promotes or inhibits) gastric emptying
Which histamine receptor is found on the basolateral membrane of the parietal cell?
T/F: In some cases of peptic ulcer disease, acid from the stomach erodes the D cells of the stomach preventing the brake mechanism for acid production.
Peptide tyrosine-tyrosine (PYY) and glucagon like peptide (GLP1 and GLP2) ____ (stimulates/inhibits) gastric emptying

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