Gastric Physiology

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Nutrients in the ileum and the colon influence gastric emptying through the _____ mechanism
P/D1 cells are located where?
The movement of Cl into the parietal cell is coupled to the movement of which ion out of the cell?
T/F: ECL cells can be activated by acetylcholine or gastrin
pespinogen and HCl are released into the lumen of the stomach... where is histamine released?
P/D1 cells release what hormone?
T/F: Rate of gastric emptying increases with rectal and colonic distention.
Peptide tyrosine-tyrosine (PYY) and glucagon like peptide (GLP1 and GLP2) ____ (stimulates/inhibits) gastric emptying
The gland of the stomach is known an ____ gland.
this hormone acts on the G cell in the antrum of the stomach to inhibit the release of gastrin
which two products of oxidative phosphorylation are important for the production of stomach acid.
Secretin ____ (promotes or inhibits) gastric emptying
Cl ions are transported into the lumen of the stomach using which type of transport protein?
these cells produce HCl and intrinsic factor
this hormone acts at the hypothalamus to suppress appetite
How does potassium get out of the parietal cell?
T/F: The parietal cell can be activated by Acetylcholine, histamine, or gastrin.
These cells in the stomach are activated by the H+ions in the stomach to reduce the release of gastrin
occurs due to a defiency in vitamin B12, which inhibits the body's ability to go through erythropoesis
Cl ions are transported into the parietal cell using which type of transport protein?
made by the I cells of the duodenum
Which histamine receptor is found on the basolateral membrane of the parietal cell?
2 causes of peptic ulcers
responsible for getting H+ ions into the lumen of the stomach
T/F: Tagemet and cimetidine prevent acid production by blocking H1 histamine receptors
2nd messenger involved in activation of the parietal cell
these cells produce pepsinogen and gastric lipase
these cells produce gastrin
___ are hormones released by the duodenum and cause feedback on stomach to decrease gastric emptying and secretions
T/F: The stomach is the major site for digestion.
CCK ____ (promotes or inhibits) gastric emptying
what activates pepsinogen?
severely delayed gastric emptying or no emptying
All stomach cell types are activated by the neurotransmitter (from the enteric nervous system) Acetylcholine, except which cell?
leptin is release by which type of tissue?
essential for binding and absorption of vitamin B12
failure of the pyloric sphincter to relax for gastric emptying... leads to projectile vomiting
prilosec or omeprazole acts by inhibiting what?
Hypoglycemia ___ (increase or decreases) gastric emptying.
T/F: In some cases of peptic ulcer disease, acid from the stomach erodes the D cells of the stomach preventing the brake mechanism for acid production.
occurs due to uncontrolled gastric emptying due to the lack of feedback inhibition by the duodenum and undigested food makes it to the colon
The superficial epithelial cells along with which other cell type in the stomach act to lubricate and protect the stomach wall?
This type of drug inhibits prostaglandin related defense mechanisms within the stomach
T/F: After a large meal, the alkaline tide (removal of bicarbonate from the parietal cell) causes an increase in the pH of the blood.
made by the S cells of the duodenum
H2 blockers prevent the binding of ____ to prevent the production of acid.
T/F: Prilosec or Omeprazole acts at the basolateral membrane of the parietal cell.
enzyme important for the production of protons and bicarbonate in the parietal cell
T/F: Large volumes in the stomach increase gastric emptying
T/F: Ileal fat increase gastric emptying.
these cells produce somatostatin
these cells produce histamine
T/F: Ghelin is released during fasting which stimulates appetite
T/F: Proton pump inhibitors bind irreversibly to the H/K proton pump.
T/F: An increase in pH (basicity) slows gastric emptying.

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Created Mar 25, 2012ReportNominate