Gastric Physiology

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Can you name the Gastric Physiology?

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what activates pepsinogen?
___ are hormones released by the duodenum and cause feedback on stomach to decrease gastric emptying and secretions
The gland of the stomach is known an ____ gland.
this hormone acts on the G cell in the antrum of the stomach to inhibit the release of gastrin
2nd messenger involved in activation of the parietal cell
T/F: Proton pump inhibitors bind irreversibly to the H/K proton pump.
P/D1 cells release what hormone?
T/F: Ileal fat increase gastric emptying.
T/F: In some cases of peptic ulcer disease, acid from the stomach erodes the D cells of the stomach preventing the brake mechanism for acid production.
these cells produce HCl and intrinsic factor
failure of the pyloric sphincter to relax for gastric emptying... leads to projectile vomiting
T/F: Large volumes in the stomach increase gastric emptying
occurs due to a defiency in vitamin B12, which inhibits the body's ability to go through erythropoesis
Secretin ____ (promotes or inhibits) gastric emptying
pespinogen and HCl are released into the lumen of the stomach... where is histamine released?
Nutrients in the ileum and the colon influence gastric emptying through the _____ mechanism
Hypoglycemia ___ (increase or decreases) gastric emptying.
Cl ions are transported into the parietal cell using which type of transport protein?
This type of drug inhibits prostaglandin related defense mechanisms within the stomach
made by the S cells of the duodenum
H2 blockers prevent the binding of ____ to prevent the production of acid.
These cells in the stomach are activated by the H+ions in the stomach to reduce the release of gastrin
T/F: The stomach is the major site for digestion.
The movement of Cl into the parietal cell is coupled to the movement of which ion out of the cell?
T/F: After a large meal, the alkaline tide (removal of bicarbonate from the parietal cell) causes an increase in the pH of the blood.
essential for binding and absorption of vitamin B12
made by the I cells of the duodenum
prilosec or omeprazole acts by inhibiting what?
QuestionAnswer
T/F: Tagemet and cimetidine prevent acid production by blocking H1 histamine receptors
2 causes of peptic ulcers
The superficial epithelial cells along with which other cell type in the stomach act to lubricate and protect the stomach wall?
these cells produce somatostatin
CCK ____ (promotes or inhibits) gastric emptying
T/F: An increase in pH (basicity) slows gastric emptying.
severely delayed gastric emptying or no emptying
leptin is release by which type of tissue?
T/F: Ghelin is released during fasting which stimulates appetite
P/D1 cells are located where?
which two products of oxidative phosphorylation are important for the production of stomach acid.
responsible for getting H+ ions into the lumen of the stomach
these cells produce gastrin
Which histamine receptor is found on the basolateral membrane of the parietal cell?
Cl ions are transported into the lumen of the stomach using which type of transport protein?
How does potassium get out of the parietal cell?
Peptide tyrosine-tyrosine (PYY) and glucagon like peptide (GLP1 and GLP2) ____ (stimulates/inhibits) gastric emptying
enzyme important for the production of protons and bicarbonate in the parietal cell
All stomach cell types are activated by the neurotransmitter (from the enteric nervous system) Acetylcholine, except which cell?
these cells produce pepsinogen and gastric lipase
T/F: The parietal cell can be activated by Acetylcholine, histamine, or gastrin.
T/F: ECL cells can be activated by acetylcholine or gastrin
T/F: Prilosec or Omeprazole acts at the basolateral membrane of the parietal cell.
these cells produce histamine
occurs due to uncontrolled gastric emptying due to the lack of feedback inhibition by the duodenum and undigested food makes it to the colon
this hormone acts at the hypothalamus to suppress appetite
T/F: Rate of gastric emptying increases with rectal and colonic distention.

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Created Mar 25, 2012ReportNominate
Tags:physiology