Russell Review

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25% of AAA will rupture if it is greater than ___cm in size.
Malignant hypertension is greater than 240/120 mmHg. It may develop in previously normotensive patients and usually superimposed on preexisting ________ hypertension.
What is the clinical result of a mural thrombus?
A person with AAA is very likely to have _________ in other vessels and has an increased risk of IHD and stroke.
At 7-28 days, the MI is _______.
Arteritis with mucocutaneous lymph node syndrome, usually occurs in children. Coronary arteries can be involved with aneurysm formation and/or thrombosis.Medium vessel vasculitis
A SEVERE fixed coronary obstruction causes this condition.
Hypertension leads to LVH and eventually myocardial __________.
Diagnosis: Destroyed internal elastic lamina and giant cells. Thickened, nodular, tender segment of temporal artery.
Necrotizing inflammation typically involving renal arteries but sparing pulmonary vessels.Medium vessel vasculitis
A fixed coronary obstruction results into this symptom.
Segmental, thrombosing, acute and chronic inflammation of medium-sized and small arteries. Tibial and radial pulses are most often affected. Occurs almost exclusively in smokers, u
Granulomatous inflammation involving the respiratory tract and necrotizing vasculitis affecting small vessels, including glomerular vessels. Associated with PR3-ANCAs. Small vessel vasculitis
Systemic vasculitis of small or medium sized muscular arteries characterized by segmental transmural necrotizing inflammation with a predilection for branch points.Medium vessel vasculitis
At 6h-7days, the MI is ______.
What is the clinical result of an occlusive thrombus?
Diagnosis: Active arteritis with destruction of tunica media by chronic inflammatory infiltrates including giant cells. Cross sections of right carortid artery at autopsy showing mLarge vessel vasculitis
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Name a manifestation of reperfusion injury.
Calcification in tunica media and not in the intima.Small muscular arteries in the pelvis, neck, and breast regions can be affected.These arterial calcifications can be seen in radiographs. Medial calcific sclerosis can also be seen involving the extremities in association with autonomic neuropathy in diabetes mellitus.
A 3-5 day old infract can have total __________ and maximal PMN infiltration.
At 29 days and beyond, the MI is _______.
Troponin I needs to be greater than _____ng/mL in order to diagnose acute MI.
Stable or Vulnerable Plaque: Thin Fibrous Cap, High lipid content, many inflammatory cells
This is the most common calcific valvular disease.Thickened cusps with dystrophic calcified plaques. Not much lipid. Not much cellular proliferation.
Necrotizing small vessel vasculitis with few or no immune deposits. Necrotizing glomerulonephritis and pulmonary capillaritis are common. Associated with MPO-ANCAs. Small vessel vasculitis
At 6h, the MI is ________.
40-50% of infarcts result from the occlusion of ______.
Eosinophil-rich granulomatous inflammation involving the respiratory tract and necrotizing vasculitis affecting small vessels. Associated with asthma and blood eosinophilia. AssociSmall vessel vasculitis
Stable or Vulnerable Plaque: Thick Fibrous Cap with low Lipid content, Few inflammatory cells
The most common form of vasculitis among elderly individuals in the United States and Europe. Therapeutic response to steroids. Unknown cause. Large vessel vasculitis
Pulseless disease. Patients less than 50yo. Granulomatous vasculitis - classically the aortic arch.
Occlusion of the LAD, Circumflex artery, and RCA result into _______ infarct.
Typically involves renal and visceral vessels but spares the pulmonary arteries. Medium vessel vasculitis

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Created Mar 23, 2011ReportNominate
Tags:review, russell