| Question | Answer |
|---|
| Thin filaments | |
| Pathology: hyperthyroidism | |
| Endocrine signaling where the molecule is secreted into the blood stream | |
| Which gland secretes TSH, FSH, LH, ACTH, GH, PRL | |
| Which gland deals with short term stress | |
| Microscopic anatomy: length of myosin fibers | |
| Which gland is connected by neuron to the hypothalmus | |
| Chemical required for muscle contraction | |
| Which gland deals with long term stress | |
| Thick filaments | |
| Endocrine signaling using gap junctions | |
| Stiffening of the body after death | |
| Which hormone lowers blood sugar | |
| Stimulation of hormone production by other hormones | |
| Microscopic anatomy: midline of myosin fiber | |
| Muscle fiber that does not require oxygen | |
| Cycle that regenerates glucose from lactic acid | |
| Pathology caused by too much GH after growth plates fuse | |
| From Smelley: How do you prevent getting type II diabetes? | |
| Effect that increases bone strength | |
| Bone forming cell | |
| Bone formation involving a cartilage intermediary; long bones | |
| | Question | Answer |
|---|
| Smallest functional contractile unit | |
| Which hormone increases calcium storage in bones | |
| Muscle fiber that requires oxygen | |
| Stimulation of hormone production by neuron | |
| Which hormone raises blood sugar | |
| Bone that has been remodeled | |
| Microscopic anatomy: distance between actin ends | |
| Endocrine signaling where the molecule exits into the lumen | |
| Endocrine signaling where the receptor is on the surface of the same producing cell | |
| Pathology: hypothyroidism in children | |
| Muscle that is striated by not voluntary; found only in the heart | |
| Endocrine signaling within the cell | |
| Muscle that is not striated and not voluntary; found in the walls of hollow visceral organs | |
| Muscle that is striated and voluntary; attaches to and covers bony skeleton | |
| Mechanosensor | |
| Neurotransmitter that tells muscles when to contract | |
| Chemical class of Melatonin, epinephrine, norepinephrine, thyroxine | |
| Pathology: resistance to insulin | |
| During contraction, the sarcomere gets shorter or remains the same | |
| Principle that bones remodel in response to physical environment | |
| Which hormone increases blood calcium | |
| During contraction, the H zone and I band gets shorter or remains the same? | |
| | Question | Answer |
|---|
| Pathology: autoimmune disorder of pancreatic islet cells | |
| Microscopic anatomy: distance between myosin fibers | |
| What happens without the above chemical | |
| Pathology: not enough GH | |
| Pathology: too much GH while still growing | |
| Bone that has never been remodeled | |
| Pathology: too much cortisone | |
| Bone destroying cell | |
| Muscle contraction without shortening of muscle | |
| Pathology: hyposecretory disorder of the adrenal cortex | |
| Chemical class of GH, Insulin, Glucagon, Insulin-like Growth Factor | |
| During contraction, the A band gets shorter or remains the same | |
| Chemical class of Testosterone, Estrogen, Progesterone, Cortisol | |
| Pathology: hypothyroid syndrome in adults | |
| Stimulation of hormone production by imbalance in the blood | |
| Bone formation direct, not involving a cartilage intermediary; flat bones | |
| Chemical class of Oxytocin, PTH, ADH | |
| Endocrine signaling where the hormone stays in the immediate interstitial fluid | |
| Groups that remodel bone | |
| Endocrine signaling that also involves neurons | |
| Muscle contraction with shortening of the muscle | |
|
1-100
Typing Challenge
Drawing a Blank
