Respiratory Pathology

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Can you name the Respiratory Diseases?

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HintDiseasePrimary Organ(s)
Spores in soil contaminated with bird or bat droppings; most involve asymptomatic-calcified hilar nodesLungs
Pneumonia that infects an entire lobeLungs
Persistent cough with copious sputum for at least 3 months for 2 consecutive years; inflammation, fibrosis, and resultant narrowing of bronchiolesLungs
Croup in children under the age of 3; loud breathing and cough with difficulty in breathingLarynx
Enlargement and destruction of air spaces distal to the terminal bronchiolesLungs
Common cold, catarrhal discharge, mucopurulent discharge secondary to bacterial infectionNose and Sinuses
Pneumonia with patchy distribution within the lobesLungs
Headache, fever, facial pain, malaise, nasal dischargeNose and Sinuses
Arteriosclerosis or increased pulmonary vascular blood flow associated with pulmonary hypertensionLungs
Often no clinical features; spread to hilar and scalene nodes, then to the adrenals, liver, brain, bone, and kidney; paraneoplastic syndromesLungs
Resistance to expiration; progressive dyspnea, reduced recoil, cyanosis, weight loss, hypoxic brain damage, bronchopneumonia, cor pulmonaleLungs
Ideopathic multisystem granulomatous process; Langhans’ or foreign body giant cells, Schaumann bodies, Asteroid bodiesLungs
Anterior bowing of the posterior wall of the maxillary sinus; dense fibrous connective tissue with myofribroblastsNose and Sinuses
Weight loss (‘consumption’), chronic bloody cough; necrotizing granulomatous inflammation, multinucleated giant cellsLungs
Benign, papillary overgrowth of epitheliumNose and Sinuses
HintDiseasePrimary Organ(s)
Later signs include ulcerations and destrunction of adjacent tissue or structures; elevated c-ANCA; “strawberry gums”Nose and Sinuses
Cough with foul-smelling mucus, fever, painLungs
Deep necrotic ulcerations; may progress to palatal perforationNose and Sinuses
Overgrowths of edematous inflammatory tissue; “Singer’s nodes”Larynx
Airless lung, appearing shrunken, red-blue, rubbery and non-crepitateLungs
Enlarged cervical nodes, unilateral serous otitis media and hearing lossNose and Sinuses
Secondary to left-sided heart disease, allergies, CNS disorders; lungs become heavy and wetLungs
95% occur from deep leg vein thrombosis; dyspnea with or without shock or sudden death, pain, hemoptysis, cor pulmonaleLungs
Sudden episodes of bronchospasm and dyspnea; Curschmann’s spirals and Charcot-Leyden crystals in sputumLungs
Associated with smoking and asbestos; pain, hemoptysis, increasing hoarsenessLarynx
Diffuse increase in permeability leading to fluid in the septa and alveolar spacesLungs
Shortly after birth, respiration becomes increasingly labored; deficiency of pulmonary surfactantLungs
Benign but aggressive neoplasm of the sinuses; inward growth of squamous epitheliumNose and Sinuses
Abnormal, permanent, and irregular dilation of bronchi associated with superimposed, necrotizing infectionLungs

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